國立台灣大學 | 生命科學院
 

 

>首頁 >師資 >鄭石通 教授兼所長 (Shih-Tong Jeng, Professor)
鄭石通 教授兼所長 (Shih-Tong Jeng, Professor)
 
職 稱 教授
出生年 1959
最高學歷 美國伊利諾大學博士
專 長 分子生物學 
E-mail stjeng@ntu.edu.tw
研究室 生命科學館 1018室
電話 02-33662538
傳真  
近年研究主題
1)甘藷傷害誘導基因的分離和性質測定
2)傷害誘導的訊息傳遞
3)植物熱干擾基因的分離和研究
研究室簡介:研究室

我們主要是以分子生物為工具去研究細胞中的基因表現和訊息傳導。近年來的研究主題為:

  1. 甘藷傷害誘導基因的分離和性質測定
    在自然環境裡,因為植物不具自由活動的能力,所以在面對眾多攝食者或微生物的侵害時,其本身自我防禦機制則扮演相當重要的抵抗和保護角色。為了釐清植物防禦系統的作用機制及探討此機制可能參與的角色與功能為何,因此本計畫在研究參與植物防禦機制的基因功能,和防禦基因被誘導的訊息傳遞過程。本實驗室已經由甘藷傷害誘導的subtraction library和differential display中,篩選出數個傷害誘導cDNA片段,並研究傷害基因所產生和誘導蛋白質的功能,和這些蛋白質在植物防禦機制上的角色。
  2. 傷害誘導的訊息傳遞
    植物受到機械性傷害或蟲咬後,會誘導一連串訊息的產生和傳遞,進而使植物防禦基因的表現,以保護植物不受到進一步傷害。實驗已經證實植物傷害後會產生H2O2、一氧化氮、和cGMP,而H2O2和一氧化氮也參與傷害誘導基因的表現。因此本計畫將進一步研究這些傷害誘導訊息的相互關係,進而瞭解傷害訊息傳導機制。同時也分析參與傷害誘導的傷害誘導啟動子,並研究可以附著在傷害誘導啟動子上的轉錄因子,以瞭解植物傷害誘導的訊息傳遞過程。
  3. 植物熱干擾基因的分離和研究
    過去二十年間,全球暖化已經造成小麥、玉米和大麥等作物每年四百萬噸的損失,估計每年損失50億美元。然而大多數研究高溫對於植物的影響,都專注於植物營養組織的熱休克誘導的部分。然而前人研究指出隨著溫度的上升,植物生殖組織,對於溫度敏感性較營養組織來的大。因此在到達熱休克反應的溫度之前,溫度的增加就已經損害了作物的產量。因此,本計劃研究中度溫度對於植物生殖組織的影響。計劃已經由生物晶片的篩選得到會受到熱干擾的基因,這些基因包括了受熱促進及受熱降低表現的基因,並且以RT-PCR進一步確認其熱干擾基因表現和溫度的相關性。希望能藉此研究可以瞭解植物中高溫干擾基因的功能及其所參與的訊息傳遞路徑,並且在全球暖化的時期,研究可以促進農作物產量的方式。

代表著作

Li, Y.-C., Wan, W.-L., Lin, J.-S., Kuo, Y.-W., King, Y.-C., Chen, Y.-C., Jeng, S.-T. * (2016) Signal transduction and regulation of IbpreproHypSys in sweet potato. Plant Cell Environ.

Wang, H.-M., To, K.-Y., Lai, H.-M., Jeng, S.-T. * (2015) Modification of flower color by suppressing β-ring carotene hydroxylase genes in Oncidium. Plant Biology.

Kuo, T. C.-Y., Chen, C.-H., Chen, S.-H., Lu, I.-H., Huang, L.-C., Lin, C.-Y., Chen, C.-Y., Lo, H.-F., Jeng, S.-T., Chen, L.-F. O. * (2015) The Effect of Red Light and Far-Red Light Conditions on Secondary Metabolism in Agarwood. BMC Plant Biology 15, 139.

Chen, C.-H., Kuo, T. C.-Y., Yang, M.-H., Chien, T.-Y., Chu, M.-., Huang, L.-C., Chen,C.-Y., Lo, H.-F., Jeng, S.-T., Chen, L.-F. O. * (2014) Identification of cucurbitacins and assembly of a draft genome for Aquilaria agallocha. BMC Genomics 15, 578.

Lin, J.-S., Lin, H.-H., Li, Y.-C., King, Y.-C., Sung, R.-J., Kuo, Y.-W., Lin, C.-C., Shen, Y.-H., Jeng, S.-T. * (2014) Carbon monoxide regulates the expression of a wound-inducible gene Ipomoelin through antioxidation and IbMAPK phosphorylation in sweet potato. Journal of Experimental Botany 65, 5279-5290.

Shen, Y.-H., Chen, Y.-H., Liu, H.-Y., Chiang, F.-Y., Wang, Y.-C., Hou, L.-Y., Lin, J.-S., Lin, C.-C., Lin, H.-H., Lai, H.-H., Jeng, S.-T. * (2014) Expression of a gene encoding β-ureidopropionase is critical for pollen germination in tomatoes. Physiologia Plantarum 150, 425-435.

Lin, J.-S., Lin, C,-C, Li,Y.-C., Wu, M.-T., Tsai, M.-H. Hsing, Y.-I. C. and Jeng, S.-T.* (2013) Interaction of small RNA–8105 and the intron of IbMYB1 RNA regulates IbMYB1 family genes through secondary siRNAs and DNA methylation after wounding. The Plant Journal 75, 781-794.

Lin, J.-S., Lin, C.-C., Lin,H.-H., Chen, Y.-C. and Jeng, S.-T.* (2012) MicroR828 regulates lignin and H2O2 accumulation in sweet potato on wounding. New Phytologist 196, 427-440.

Chang, W. C., Liu, K. L., Hsu, F. C., Jeng, S. T., Cheng, Y. S. * (2012) Ipomoelin, a jacalin-related lectin with a compact tetrameric association and versatile carbohydrate binding properties regulated by its N terminus. PLoS ONE 7(7):e40618.

Lin, C.-C., Jih, P-J, Lin, H.-H., Lin, J.-S., Chang, L.-L., Shen, Y.-H., Jeng, S.-T. (2011) Nitricoxide activates superoxide dismutase and ascorbate peroxidase to repress the cell death induced by wounding. Plant Molecular Biology 77, 235-249

Lin, C.-C., Chu, C.-F., Liu, P.-H., Lin, H.-H., Liang, S.-C., Hsu, W.-E., Lin, J.-S., Wang, H.-M., Chang, L.-L., Chien, C.-T., Jeng, S.-T. (2011) Expression of an Oncidium Gene Encoding a Patatin-Like Protein Delays Flowering in Arabidopsis by Reducing Gibberellin Synthesis. Plant and Cell Physiology 52, 421-435

Lin, H.-H., Huang, L.-F., Su, H.-C. Jeng, S.-T. (2009) Effects of the multiple polyadenylation signal AAUAAA on mRNA 30-end formation and gene expression. Planta 230, 699-712.

Chen, Y.-C., Lin, H.-H. and Jeng, S.-T. (2008) Calcium influxes and mitogen-activated protein kinase kinase activation mediate ethylene inducing ipomoelin gene expression in sweet potato. Plant, Cell and Environments 31, 62-72.
Chen, H.-F. Chang, M.–H., Chiang, B.-L. Jeng, S.-T. (2006) Oral immunization of mice using transgenic tomato fruit expressing VP1 protein from enterovirus. Vaccine 24, 2944-2951.

Chen ,Y.-C., Chang, H.-S., Lai, H.-M. and Jeng, S.-T. (2005) Characterization of the wound-inducible protein ipomoelin from sweet potat. Plant, Cell and Environments 28, 251-259.

Liu, Y.-C., Lin, S.-H., Lai, H.-M., and Jeng, S.-T. (2005) Detection of Genetically modified soybean and its product tou-kan by polymerase chain reaction with dual pairs of DNA primers. European Food Research and Technology 221, 725-730.

Jih, P-J, Chen, Y-C and Jeng, S.-T. (2003) Involvement of Hydrogen Peroxide and Nitric Oxide in Expression of the Ipomoelin Gene from Sweet Potato. Plant Physiology 132, 1-9.

Chen, Y.-C., Tseng, B.-W., Huang, Y.-L. Liu, Y.-C. and Jeng, S.-T. (2003) Expression of the ipomoelin gene from sweet potato is regulated by dephosphorylated proteins, calcium ion, and ethylene. Plant, Cell and Environments 26, 1373-1383.

開設課程
  分子生物學
  生物學
  生物化學
  細胞生物學
  生物技術核心實驗
實驗室生活

Copyright © 2004, 國立台灣大學植物科學研究所

最後更新: